An article in the Journal of the American College of Cardiology notes that evidence of cardiovascular damage was found in 25% of hospitalized coronavirus patients.
Studies have found elevated enzyme levels and other signs that indicate damage to the heart, even in patients who have not previously experienced heart muscle problems. It is not known whether this damage is permanent and whether the virus can cause permanent dysfunction of an organ normally functioning before the disease.
Different types of structural damage have been identified that may be associated with the onset of myocarditis in a coronavirus survivor.
The results of the study provide an opportunity to consider new methods that can help doctors better understand the mechanism of damage to the cardiovascular system, which will allow faster identification of patients at risk.
In the research work, first of all, it was taken into account whether there is an increase in the cardiac regulatory protein troponin, which is responsible for the contraction of the heart muscle, in combination with the presence of cardiographic abnormalities. Indeed, if both risk factors are found in one patient, such a patient is most vulnerable to infection.
In some people, because COVID-19 decreases lung function, it can deprive the heart of adequate oxygen. This sometimes triggers an inflammatory response that puts a lot of strain on the heart as the body is fighting infection.
The virus can also invade blood vessels or cause inflammation inside them, leading to blood clots. Doctors are going to fight this problem by prescribing blood thinners, if necessary.